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Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components. / Lindner, Paula; Christensen, Soren Brogger; Nissen, Poul; Moller, Jesper Vuust; Engedal, Nikolai.
I:
Cell Communication and Signaling, Bind 18, 12, 2020.
Publikation: Bidrag til tidsskrift › Tidsskriftartikel › Forskning › fagfællebedømt
Harvard
Lindner, P
, Christensen, SB, Nissen, P, Moller, JV & Engedal, N 2020, '
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components',
Cell Communication and Signaling, bind 18, 12.
https://doi.org/10.1186/s12964-019-0499-zAPA
Lindner, P.
, Christensen, S. B., Nissen, P., Moller, J. V., & Engedal, N. (2020).
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components.
Cell Communication and Signaling,
18, [12].
https://doi.org/10.1186/s12964-019-0499-zVancouver
Lindner P
, Christensen SB, Nissen P, Moller JV, Engedal N.
Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components.
Cell Communication and Signaling. 2020;18. 12.
https://doi.org/10.1186/s12964-019-0499-zAuthor
Lindner, Paula ; Christensen, Soren Brogger ; Nissen, Poul ; Moller, Jesper Vuust ; Engedal, Nikolai. / Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components. I: Cell Communication and Signaling. 2020 ; Bind 18.
Bibtex
@article{cbe9d3d952814172826e63d8f898dbcc,
title = "Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components",
keywords = "Thapsigargin, SERCA, Unfolded protein response, DR5, Caspase-8, PERK, ATF4, CHOP, IRE1, XBP1s, JNK, LC3B, Cell death, Apoptosis, Autophagy",
author = "Paula Lindner and Christensen, {Soren Brogger} and Poul Nissen and Moller, {Jesper Vuust} and Nikolai Engedal",
year = "2020",
doi = "10.1186/s12964-019-0499-z",
language = "English",
volume = "18",
journal = "Cell Communication and Signaling",
issn = "1478-811X",
publisher = "BioMed Central Ltd.",
}
RIS
TY - JOUR
T1 - Cell death induced by the ER stressor thapsigargin involves death receptor 5, a non-autophagic function of MAP1LC3B, and distinct contributions from unfolded protein response components
AU - Lindner, Paula
AU - Christensen, Soren Brogger
AU - Nissen, Poul
AU - Moller, Jesper Vuust
AU - Engedal, Nikolai
PY - 2020
Y1 - 2020
KW - Thapsigargin
KW - SERCA
KW - Unfolded protein response
KW - DR5
KW - Caspase-8
KW - PERK
KW - ATF4
KW - CHOP
KW - IRE1
KW - XBP1s
KW - JNK
KW - LC3B
KW - Cell death
KW - Apoptosis
KW - Autophagy
U2 - 10.1186/s12964-019-0499-z
DO - 10.1186/s12964-019-0499-z
M3 - Journal article
C2 - 31987044
VL - 18
JO - Cell Communication and Signaling
JF - Cell Communication and Signaling
SN - 1478-811X
M1 - 12
ER -
